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http://repositorio.inger.gob.mx/jspui/handle/20.500.12100/17287
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Campo DC | Valor | Lengua/Idioma |
---|---|---|
dc.rights.license | http://creativecommons.org/licenses/by/4.0 | es_MX |
dc.creator | ARMANDO LUNA LOPEZ | es_MX |
dc.creator | VIRIDIANA YAZMIN GONZALEZ PUERTOS | es_MX |
dc.creator | Jacqueline Romero Ontiveros | es_MX |
dc.creator | JOSE LUIS VENTURA GALLEGOS | es_MX |
dc.creator | ALEJANDRO ZENTELLA DEHESA | es_MX |
dc.creator | LUIS ENRIQUE GOMEZ QUIROZ | es_MX |
dc.creator | MINA KONIGSBERG FAINSTEIN | es_MX |
dc.date | 2013 | - |
dc.date.accessioned | 2021-10-28T18:06:49Z | - |
dc.date.available | 2021-10-28T18:06:49Z | - |
dc.identifier.uri | http://repositorio.inger.gob.mx/jspui/handle/20.500.12100/17287 | - |
dc.description | Cells can respond to damage and stress by activating various repair and survival pathways. One of these responses can be induced by preconditioning the cells with sublethal stress to provoke a prosurvival response that will prevent damage and death, and which is known as hormesis. Bcl-2, an antiapoptotic protein recognized by its antioxidant and prosurvival functions, has been documented to play an important role during oxidative-conditioning hormesis. Using an oxidative-hormetic model, which was previously established in the L929 cell line by subjecting the cells to a mild oxidative stress of 50 μM H₂O₂ for 9 h, we identified two different transductional mechanisms that participate in the regulation of Bcl-2 expression during the hormetic response. These mechanisms converge in activating the nuclear transcription factor NF-κB. Interestingly, the noncanonical p50 subunit of the NF-κB family is apparently the subunit that participates during the oxidative-hormetic response. | es_MX |
dc.format | Adobe PDF | es_MX |
dc.language | eng | es_MX |
dc.publisher | Elsevier | es_MX |
dc.relation | https://www.sciencedirect.com/science/article/abs/pii/S0891584913001913?via%3Dihub#! | es_MX |
dc.relation.requires | Si | es_MX |
dc.rights | Acceso Abierto | es_MX |
dc.source | Free Radical Biology and Medicine (0891-5849) Vol. 63 (2013) | es_MX |
dc.subject | BIOLOGÍA Y QUÍMICA | es_MX |
dc.subject | Ciencias de la vida | es_MX |
dc.subject | Biología celular | es_MX |
dc.subject | Químicos inorgánicos | es_MX |
dc.subject | Radicales libres | es_MX |
dc.subject | Estrés oxidativo | es_MX |
dc.subject | Hormesis | es_MX |
dc.subject | Respuesta adaptativa | es_MX |
dc.subject | Cell biology | es_MX |
dc.subject | Inorganic chemicals | es_MX |
dc.subject | Free radicals | es_MX |
dc.subject | Oxidative stress | es_MX |
dc.subject | Adaptative response | es_MX |
dc.title | A noncanonical NF-κB pathway through the p50 subunit regulates Bcl-2 overexpression during an oxidative-conditioning hormesis response | es_MX |
dc.type | Artículo | es_MX |
dc.audience | Researchers | es_MX |
dc.creator.id | LULA690630HDFNPR02 | es_MX |
dc.creator.id | GOPV820119MDFNRR03 | es_MX |
dc.creator.id | CA1219043 | es_MX |
dc.creator.id | VEGL670518HGRNLS01 | es_MX |
dc.creator.id | ZEDA570306HDFNHL05 | es_MX |
dc.creator.id | GOQL691127HMSMRS00 | es_MX |
dc.creator.id | KOFM650625MDFNNN02 | es_MX |
dc.creator.nameIdentifier | curp | es_MX |
dc.creator.nameIdentifier | curp | es_MX |
dc.creator.nameIdentifier | ca | es_MX |
dc.creator.nameIdentifier | curp | es_MX |
dc.creator.nameIdentifier | curp | es_MX |
dc.creator.nameIdentifier | curp | es_MX |
dc.creator.nameIdentifier | curp | es_MX |
Aparece en las colecciones: | 1. Artículos |
Ficheros en este ítem:
Fichero | Descripción | Tamaño | Formato | |
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Free Radical Biology and Medicine (0891-5849) Vol. 63 (2013).pdf | 2.02 MB | Adobe PDF | Visualizar/Abrir |